Portuguese
corpo do estômago 1
CT failed to demonstrate two fundal and 19 antral or gastric body lesions.
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Results: In the gastric body, the densities of H. pylori were not different among groups.
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Upper gastrointestinal endoscopy revealed irregularly elevated tumors in the lower anterior gastric body, which were diagnosed to be metastasis from HCC.
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Gastroendoscopic findings showed an 8.0-cm submucosal tumor with ulcer on the greater curvature of the gastric body.
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The upper posterior gastric body could be the most feasible location for performing TU-ESD, perhaps because of the difference in the subcutaneous dissection time.
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Seventy-two hours later, gastric corpus tissues were taken for quantitative histological assessment.
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Apoptosis was also significantly increased in gastric corpus biopsies obtained from H. pylori-infected humans with moderate degrees of hypergastrinemia.
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Design: Epithelial cells were isolated from the gastric corpus of healthy stomachs and stomachs with drug-induced and inflammation-induced SPEM lesions.
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The nitric oxide synthase (NOS) activity was measured in the surface and deep mucosa of the gastric corpus.
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We conclude that hypergastrinemia specifically renders cells within the gastric corpus epithelium more susceptible to induction of apoptosis by radiation or H. pylori.
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Conclusions: Gastric acid plays a role in suppressing the proximal propagation of H. pylori-induced gastritis to the body of stomach.